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2023 ESC Guidelines for Acute Coronary Syndromes

Clinical Quick Reference — STEMI, NSTEMI, and Unstable Angina Management

Published: European Heart Journal (2023)
Societies: European Society of Cardiology (ESC)
DOI: 10.1093/eurheartj/ehad191
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What's New (2023 vs. 2020)

Topic Key Change COR
CCTA in NSTE-ACS Now recommended as part of initial imaging workup for patients with suspected NSTE-ACS (previously optional) 2a
Early invasive strategy timing Expanded to include GRACE risk score >140 as indication for within 24h invasive approach 1
DAPT in high bleeding risk (HBR) Options for shortened (3–6 mo) or extended DAPT; single antiplatelet after 1 mo in specific HBR scenarios 2b
CABG DAPT resumption If DAPT stopped for CABG, resume for remainder of 12-month period 1
Cancer patients with ACS Invasive strategy recommended if expected survival ≥6 months; conservative approach if <6 months 1
Cardiac arrest management Evaluate neurological status; consider delayed angiography; temperature control ≥37.7°C; early device implantation if needed 1

Initial Assessment & Diagnosis

Clinical Presentation: Think "A.C.S."

Rapid Assessment Framework

A — Abnormal ECG? 12-lead ECG within 10 min of first medical contact. Assess for ST-elevation, ST-depression, T-wave inversion, or other ischemic changes. 1
C — Clinical Context? Chest pain character (pressure, tightness, burning), duration, associated symptoms (dyspnea, diaphoresis, nausea). Vital signs, risk factors, prior cardiovascular history.
S — Stable Patient? Assess hemodynamic stability, signs of cardiogenic shock, life-threatening arrhythmias, and mechanical complications.

High-Sensitivity Cardiac Troponin (hs-cTn)

Sampling times: Blood at 0 h and 1 h (or 0 h and 2–3 h depending on assay). ESC 0/1h rule-in/rule-out algorithms have >99% sensitivity and NPV ≥99%. 1

Central lab preferred: Central laboratory assays have higher sensitivity than point-of-care (POC) tests. POC assays may miss 20–30% of early MI. 1

Clinical variables: Age, renal function, elevated hs-cTn baseline in healthy older adults. Use 0/3h algorithm if hs-cTn negative and no ongoing symptoms or 1h/2h results are inconclusive.

ECG Interpretation

12-lead ECG recorded immediately and interpreted by trained personnel. Repeat if symptoms persist or recur.

ECG Finding Working Diagnosis Action
ST elevation ≥0.1 mV in ≥2 contiguous leads (or new LBBB) STEMI Immediate reperfusion: primary PCI <120 min or fibrinolysis. Door-to-balloon <120 min.
ST-depression, T-wave inversion, biphasic T, pseudonormalization of T NSTE-ACS (high likelihood) Serial hs-cTn; early invasive strategy if high-risk features; GRACE score for risk assessment.
Normal ECG UA or early ACS Repeat ECG if symptoms persist. Serial hs-cTn at 0/1 h; if inconclusive, repeat at 3 h. HEART score risk stratification.
Pearl: Serial hs-cTn with the 0/1h algorithm allows safe, rapid rule-out of MI in <2 hours with >99% sensitivity. Dynamic ECG changes (especially transient ST elevation) suggest high ischemic risk and warrant early invasive strategy.

Risk Stratification

GRACE Score for NSTE-ACS

Calculate using the GRACE ACS Risk Calculator or GRACE In-Hospital Calculator. GRACE >140 indicates high risk and warrants early invasive strategy within 24 h. 1

HEART Score for Chest Pain

Use the HEART Score Calculator to rapidly stratify chest pain patients into low (<2% 30-day MACE), intermediate, or high-risk categories. Useful in primary care and ED settings.

Very High-Risk Features (Immediate Invasive Angiography)

Feature Action
Haemodynamic instability or cardiogenic shock Emergency angiography + revascularization; consider mechanical support (IABP, MCS)
Recurrent or refractory chest pain despite medical therapy Emergency angiography within hours
Life-threatening arrhythmias (VF, sustained VT) or cardiac arrest during hospitalization Emergency angiography after resuscitation; temperature control ≥37.7°C; consider ICU/mechanical support
Mechanical complications (papillary muscle rupture, VSD, free wall rupture) Emergency angiography followed by surgical consultation

High-Risk Features (Early Invasive Strategy within 24 h)

NSTE-ACS Management Strategy

Invasive Strategy Timing

Risk Category Timing Recommendation COR
Very high-risk (haemodynamic instability, recurrent chest pain, arrhythmias, mechanical complications) Immediate angiography 1
High-risk NSTEMI (hs-cTn elevation, GRACE >140, dynamic ECG) Early invasive within 24 h from diagnosis 1
Intermediate-risk NSTE-ACS (confirmed NSTEMI, no very high-/high-risk features) Early invasive strategy within 24 h; selective approach if no high-risk criterion 2a
Low-risk NSTE-ACS (HEART score 0–3, serial hs-cTn negative) Selective invasive approach; non-invasive stress testing acceptable 1

Complete vs. Culprit-Only Revascularization

1 Complete revascularization (within 45 days) recommended in hemodynamically stable NSTEMI patients with multivessel disease. Can be performed at index PCI or staged.

2a Culprit-only PCI acceptable in hemodynamically unstable patients or those with extensive myocardial necrosis.

STEMI Management & Reperfusion Strategy

Reperfusion Strategy Selection

Primary PCI vs. Fibrinolysis

Primary PCI (PPCI): 1 Preferred if performed within 120 minutes of ECG-based diagnosis in a PCI-capable centre. Door-to-balloon <120 min. Includes catheter-based angiography ± PCI with balloon, stent, or other devices.
Fibrinolytic therapy: 1 Recommended within 12 h of symptom onset if: (1) PCI not available within 120 min, (2) early presentation (<3 h), (3) contraindications to PPCI. Administer within 30 min of first medical contact (door-to-needle <30 min).
Rescue PCI: 1 Indicated if fibrinolysis fails (ST-segment resolution <50% at 60–90 min, worsening ischemia, or ongoing chest pain). Transfer to PCI centre for emergency angiography + intervention.
Routine early PCI after successful fibrinolysis: 2a Angiography ± PCI between 2–24 h post-fibrinolysis recommended for risk stratification and culprit vessel assessment.

Time Targets

Door-to-balloon (PPCI): <120 minutes from first medical contact to balloon inflation. 1

Door-to-needle (fibrinolysis): <30 minutes from hospital arrival to fibrinolytic administration. 1

System delay (EMS to PPCI): <60 minutes for single-centre systems; <90 minutes for regional STEMI networks with transfers.

Contact-to-balloon (pre-hospital ECG + PPCI): <120 minutes total system time.

Fibrinolytic Agents (if PPCI not available)

Agent Dose Route
Tenecteplase (TNK) Weight-adjusted 30–50 mg Single i.v. bolus
Alteplase (rt-PA) 15 mg bolus, then 0.75 mg/kg over 30 min (max 50 mg), then 0.5 mg/kg over 60 min (max 35 mg) i.v. infusion
Reteplase 10 U bolus, repeat 10 U after 30 min i.v. bolus
Pitfall: System delays > 120 min door-to-balloon. Establish PCI networks with rapid door-to-EMS notification, rapid transfer protocols, and pre-hospital ECG transmission. Even if PPCI >120 min, it may still be superior to fibrinolysis in some high-risk scenarios.

Antithrombotic Therapy

Dual Antiplatelet Therapy (Aspirin + P2Y₁₂ Inhibitor)

Agent Loading Dose Maintenance Duration
Aspirin 150–300 mg oral, or 75–250 mg i.v. 75–100 mg/day Lifelong
Ticagrelor 180 mg oral 90 mg b.i.d. 12 months (default); shortened if HBR
Prasugrel 60 mg oral (30 mg if <60 kg or ≥75 y) 5–10 mg/day 12 months (default); shortened if HBR
Clopidogrel 300–600 mg oral 75 mg/day 12 months if prasugrel/ticagrelor unavailable or contraindicated

1 All ACS patients should receive aspirin + P2Y₁₂ inhibitor for ≥12 months. Prasugrel or ticagrelor preferred over clopidogrel in ACS undergoing PCI.

Anticoagulation in ACS (During Acute Phase & Procedures)

Agent Dosing Key Features
UFH (Unfractionated Heparin) Bolus: 70–100 U/kg i.v.; Infusion: 1.5 U/kg/h; target aPTT 50–70 s Standard for STEMI + PPCI. Short half-life. Monitor aPTT. HIT risk.
Enoxaparin Loading: 0.5 mg/kg s.c.; Maintenance: 0.1 mg/kg s.c. b.i.d. until stabilization Subcutaneous; no monitoring. Alternative to UFH. Renal dosing if CrCl <30.
Bivalirudin Bolus: 0.75 mg/kg i.v.; Infusion: 1.75 mg/kg/h for ≤4 h (PCI setting) Direct thrombin inhibitor. No monitoring. Lower HIT risk. Alternative to UFH at PPCI.
Fondaparinux 2.5 mg s.c. daily (<50 kg: 1.5 mg; >100 kg: 10 mg) Anti-Xa. Preferred for NSTE-ACS non-invasive management. Caution if PCI <24 h (stent thrombosis risk).

✓ DO

  • Give aspirin + P2Y₁₂ inhibitor to all ACS patients regardless of management strategy. 1
  • Combine antiplatelet therapy with anticoagulation at PCI for all ACS patients. 1
  • Choose ticagrelor or prasugrel (not clopidogrel) for ACS undergoing PCI. 1
  • Use UFH or bivalirudin at PPCI; enoxaparin or fondaparinux for medical management.
  • Assess bleeding risk using HAS-BLED or ARC-HBR scores to guide DAPT intensity.

✗ DO NOT

  • Use aspirin monotherapy for ACS. 3
  • De-escalate antiplatelet therapy in first 30 days post-ACS. 3
  • Combine two anticoagulants routinely (e.g., UFH + enoxaparin) unless transitioning agents. 3
  • Use gemfibrozil with any statin (prohibitively high myopathy risk).

Dual Antiplatelet Therapy (DAPT) Duration

Standard DAPT Duration: 12 Months

1 Default: 12 months DAPT (aspirin + P2Y₁₂ inhibitor) for all ACS patients, regardless of stent type or clinical presentation.

After 12 months, continue aspirin indefinitely. P2Y₁₂ inhibitor discontinued (unless indication for extended therapy based on ischemic/bleeding risk).

Shortened DAPT (<12 months)

Scenario Duration COR
High bleeding risk (HBR) patients; event-free after 3–6 mo DAPT 3–6 months DAPT 2a
Patients on OAC requiring DAPT; switch to single antiplatelet + OAC 1 month DAPT, then aspirin + OAC (or OAC alone) 1

Extended DAPT (>12 months)

2a In patients event-free after 12 months DAPT with high ischemic risk (prior MI, multivessel disease) and low bleeding risk, extending DAPT with a P2Y₁₂ inhibitor may be considered for long-term secondary prevention.

Use the DAPT Score Calculator to guide DAPT duration decisions based on ischemic risk (prior MI, diabetes, age) vs. bleeding risk (age, CrCl, hemoglobin).

Secondary Prevention & Long-Term Management

Antiplatelet & Anticoagulation Therapy

1 Lifelong aspirin (75–100 mg/day) for all patients with established coronary artery disease or prior ACS.

1 P2Y₁₂ inhibitor for 12 months post-ACS; beyond 12 months based on ischemic vs. bleeding risk balance using DAPT Score.

Lipid-Lowering Therapy

1 High-intensity statin: Atorvastatin ≥80 mg or rosuvastatin ≥20 mg daily for all ACS patients.

LDL-C target: <55 mg/dL (<1.4 mmol/L). Add ezetimibe (10 mg/day) if LDL-C ≥55 on statin monotherapy.

PCSK9 inhibitors: Consider if LDL-C remains ≥55 mg/dL despite statin + ezetimibe (evolocumab, alirocumab, inclisiran).

Blood Pressure Management

Target: <130/80 mmHg. 1 ACE inhibitor or ARB indicated for post-MI patients with LVEF ≤40% or diabetes, regardless of BP.

Beta-blockers: Recommended for ≥3 years post-MI (especially if LVEF <40%), or indefinitely if recurrent ischemia or arrhythmia.

Lifestyle & Cardiac Rehabilitation

✓ DO

  • Enroll in comprehensive cardiac rehabilitation (≥3 months). 1
  • Smoking cessation — absolute priority. 1
  • Aerobic exercise ≥150 min/week moderate intensity (or 75 min high intensity).
  • Mediterranean or DASH diet pattern.
  • Weight management: BMI 18.5–24.9 kg/m².
  • Consider SGLT2 inhibitors if diabetes or heart failure (reduced ejection fraction).

Revascularization Strategy

PCI vs. CABG in Multivessel Disease

PCI: 1 Preferred initial strategy for STEMI (primary PCI <120 min). For NSTEMI with multivessel disease, PCI is standard if anatomy suitable.

CABG: Consider in left main stenosis, complex 3-vessel disease, or if PCI failure/contraindication. 2a CABG timing: elective after stabilization, preferably after completing DAPT (if not urgent).

Complete vs. Culprit-Only Revascularization

1 Complete revascularization recommended in hemodynamically stable NSTEMI/ACS patients with multivessel disease. Timing: index procedure or staged within 45 days (with PCI of non-culprit lesions).

2a Culprit-only PCI may be used if hemodynamic instability, extensive necrosis, or other factors preclude safe complete revascularization.

DAPT After CABG

1 DAPT resumption after CABG: If DAPT was stopped for CABG surgery, resume DAPT for the remainder of the 12-month ACS period (unless absolute HBR contraindication).

Acute Complications Management

Cardiogenic Shock (CS)

Definition: Systolic BP <90 mmHg for ≥30 min (or requiring vasopressor/inotropic support) due to inadequate cardiac output despite adequate/elevated filling pressures.

1 Early revascularization (PCI or CABG): Strongly recommended. Percutaneous mechanical circulatory support (IABP, extracorporeal membrane oxygenation [ECMO], or other devices) as bridge to definitive therapy. 1

Pharmacotherapy: Vasopressors (norepinephrine preferred) + inotropes (milrinone, dobutamine); optimize filling pressures; vasodilators if elevated afterload.

Mechanical Complications

Complication Clinical Clues Diagnostic Confirmation Management
Acute mitral regurgitation (papillary muscle rupture) Acute pulmonary edema, hypotension, new holosystolic murmur, prominent V wave on PA catheter Echocardiography: flail leaflet, ruptured papillary muscle Emergency surgery after hemodynamic stabilization; vasodilators (nitroprusside) as bridge
Ventricular septal defect (VSD) Acute pulmonary edema, hypotension, palpable thrill, systolic murmur at LSB, elevated RV saturation on oximetry Echocardiography: septal discontinuity + left-to-right shunt; cardiac catheterization: step-up in saturation at RV Emergency surgery after stabilization; IABP/mechanical support
Free wall rupture Sudden cardiogenic collapse, electromechanical dissociation, cardiac tamponade Echo: pericardial fluid + pseudoaneurysm or rupture site Emergency surgery; aggressive resuscitation with fluids + vasopressors pending OR

Arrhythmias Post-ACS

Atrial fibrillation: 2a Rate control (beta-blocker, non-dihydropyridine calcium channel blocker) preferred acutely. OAC indicated for CHA₂DS₂-VASc ≥1 in women or ≥2 in men (use CHA₂DS₂-VASc Calculator). Triple therapy (aspirin + P2Y₁₂ inhibitor + OAC) if both DAPT and OAC indicated.

Ventricular arrhythmias (VT/VF): 1 Amiodarone or other antiarrhythmics for hemodynamically significant rhythms. ICD placement if LVEF ≤35% after 40 days post-MI and optimal medical therapy.

Pitfall: Delaying mechanical revascularization in cardiogenic shock or mechanical complications. Early coronary angiography + revascularization are critical for outcomes. Manage hemodynamically while arranging emergency procedures.

Special Populations

Elderly Patients (≥75 years)

ACS management follows same principles as younger patients. Assess frailty, cognitive status, and comorbidities (CKD, anemia, prior bleeding).

1 Invasive strategy: Recommended based on functional status and clinical presentation, not age alone.

2a DAPT duration: Consider shortened DAPT (6–9 mo) in elderly with high bleeding risk and low ischemic risk.

Prasugrel dosing: Reduce maintenance to 5 mg/day if ≥75 years (instead of standard 10 mg).

Chronic Kidney Disease

Estimate renal function using CKD-EPI eGFR or Cockcroft-Gault CrCl for drug dosing.

CKD Stage eGFR Drug Adjustments for ACS
Stage 3a–3b 30–59 mL/min/1.73m² No major adjustments. Monitor renal function and potassium.
Stage 4 15–29 mL/min/1.73m² Enoxaparin: caution (increased bleeding risk). Fondaparinux: avoid. Monitor renal function closely.
Stage 5 (ESRD) <15 mL/min/1.73m² UFH preferred over LMWH. Avoid fondaparinux. Prasugrel caution (consider reduced loading/maintenance). Dialyze before elective procedures.

Diabetes Mellitus

1 Early invasive strategy: Recommended for diabetic ACS patients. All should receive high-intensity statin therapy and LDL-C <55 mg/dL target.

SGLT2 inhibitor: Consider if reduced LVEF or heart failure to improve outcomes (empagliflozin, dapagliflozin).

Cancer Patients with ACS

1 Expected survival ≥6 months: Treat ACS with standard strategies (PCI if appropriate, dual antiplatelet therapy, high-intensity statin).

1 Expected survival <6 months: Conservative medical management may be considered, but should not automatically exclude invasive therapy if patient desires aggressive approach.

Prior CABG

1 DAPT resumption after CABG: If DAPT was stopped pre-operatively, resume for ≥12 months total ACS duration (unless HBR). Consider allograft-related complications (SVG patency, progression of native CAD).

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